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The CrossFit stimulus—constantly varied high-intensity functional movement coupled with meat and vegetables, nuts and seeds, some fruit, little starch, and no sugar—prepares you for the demands of a healthy, functional, independent life and provides a hedge against chronic disease and incapacity. This stimulus is elegant in the mathematical sense of being marked by simplicity and efficacy. The proven elements of this broad, general, and inclusive fitness, in terms of both movement and nutrition, are what we term our CrossFit Essentials.

Dr. Michael Eades argues insulinocentric bias has kept most researchers focused on using insulin over the past 90 years of Type 1 diabetes treatment. The problem, he claims, is that the treatment has proven largely ineffective at getting and keeping patients’ blood glucose levels in normal ranges. Citing a landmark study by Dr. Robert Unger, Eades observes that glucagon, not insulin, may be the primary hormone causing the metabolic chaos seen in the disease. But despite the promise of alternative T1DM treatments emerging in clinical studies with animals, mainstream medicine continues to pursue insulin pumps and an ever-expanding array of injectable insulins and insulin analogs.

Read MoreA Different Perspective on Treatment of Type 1 Diabetes

Dr. Malcolm Kendrick offers a brief history of Type 2 diabetes, from references to the disease by ancient Roman physicians, through a well-known 17th-century discourse, and into the 20th century, when a shift in eating habits corresponded to a sudden rise in diabetes incidence. Kendrick observes that the shift occurred as the diet-heart hypothesis, the belief that consuming saturated fat raises cholesterol levels and promotes heart disease, became more widely accepted. As the dietary guidelines changed, people began consuming less fat and more carbohydrates — to devastating effect.

Read MoreThe diet-heart hypothesis, part 5 — Impact on Type 2 diabetes

This 2015 trial used Womens’ Health Study data and a consideration of three alternative lipid biomarkers to demonstrate that LDL cholesterol markers significantly under- or overestimate heart disease risk in a meaningful segment of the population. Specifically, in individuals with low levels of insulin resistance — such as those following a low-carb diet alongside regular exercise — high LDL-C levels may not indicate elevated heart disease risk. Conversely, insulin-resistant individuals with low LDL-C levels may still be at a high risk of heart disease.

Read MoreDiscordance of Low-Density Lipoprotein (LDL) cholesterol with alternative LDL-related measures and future coronary events

In this brief 2019 review, Robert DuBroff and Michel de Lorgeril argue existing evidence fails to support the diet-heart hypothesis — that is, the hypothesis that saturated fat increases cholesterol and, in the process, heart disease risk. If the diet-heart hypothesis were true, reducing saturated fat intake should lower cholesterol and reduce heart disease risk and mortality. However, DuBroff and de Lorgeril find the majority of the available evidence ultimately fails to support the hypothesis.

Read MoreFat or fiction: The diet-heart hypothesis

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